This study examined the role of the high-affinity copper uptake protein hCTR1 in cellular copper homeostasis and found hCTR1 was internalized in response to raised copper levels. The work from this thesis supports a model in which the regulation of hCTR1 is partially or wholly dependent upon internal copper levels
Notes
Submitted to the School of Life and Environmental Sciences of the Faculty of Science and Technology, Deakin University
Degree conferred 2007
Thesis (Ph.D.)--Deakin University, Victoria, 2006
Bibliography
Includes bibliographical references (leaves 210-219)
