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Complaints (Criminal procedure) -- See Also the narrower term Informers


  1
 

Complaints (Retail trade) -- See Consumer complaints


  1
Complaints (Rhetoric) : Literature and complaint in England, 1272-1553 / Wendy Scase  2007 1
Complaints (Rhetoric) -- In literature. : Complaint : grievance among friends / Avital Ronell  2018 1
Complaints (Rhetoric) -- Juvenile fiction. : Don't let the pigeon drive the bus! / words and pictures by Mo Willems  2003 1
Complaints (Rhetoric) -- Philosophy. : Complaint : grievance among friends / Avital Ronell  2018 1
Spenser, Edmund, 1552?-1599. Complaints : The new poet : novelty and tradition in Spenser's Complaints / Richard Danson Brown  1999 1
 

Complancha -- See Laments


  1
Walton, Izaak, 1593-1683. Compleat angler. : The compleat angler, 1653-1676 / Izaak Walton ; edited, with an introduction and commentary by Jonquil Bevan  1983 1
  Complement -- 2 Related Subjects   2
 

Complement 2 -- See Complement C2


A component of the CLASSICAL COMPLEMENT PATHWAY. C2 is cleaved by activated COMPLEMENT C1S into COMPLEMENT C2B and COMPLEMENT C2A. C2a, the COOH-terminal fragment containing a SERINE PROTEASE, combines with COMPLEMENT C4B to form C4b2a (CLASSICAL PATHWAY C3 CONVERTASE) and subsequent C4b2a3b (CLASSICAL PATHWAY C5 CONVERTASE)
  1
 

Complement 3 -- See Complement C3


A glycoprotein that is central in both the classical and the alternative pathway of COMPLEMENT ACTIVATION. C3 can be cleaved into COMPLEMENT C3A and COMPLEMENT C3B, spontaneously at low level or by C3 CONVERTASE at high level. The smaller fragment C3a is an ANAPHYLATOXIN and mediator of local inflammatory process. The larger fragment C3b binds with C3 convertase to form C5 convertase
  1
 

Complement 3 Convertase, Alternative Pathway -- See Complement C3 Convertase, Alternative Pathway


A serine protease that is the complex of COMPLEMENT C3B and COMPLEMENT FACTOR BB. It cleaves multiple COMPLEMENT C3 into COMPLEMENT C3A (anaphylatoxin) and COMPLEMENT C3B in the ALTERNATIVE COMPLEMENT ACTIVATION PATHWAY
  1
 

Complement 3a -- See Complement C3a


The smaller fragment generated from the cleavage of complement C3 by C3 CONVERTASE. C3a, a 77-amino acid peptide, is a mediator of local inflammatory process. It induces smooth MUSCLE CONTRACTION, and HISTAMINE RELEASE from MAST CELLS and LEUKOCYTES. C3a is considered an anaphylatoxin along with COMPLEMENT C4A; COMPLEMENT C5A; and COMPLEMENT C5A, DES-ARGININE
  1
 

Complement 3b-Associated Factor B-fragment Bb -- See Complement C3 Convertase, Alternative Pathway


A serine protease that is the complex of COMPLEMENT C3B and COMPLEMENT FACTOR BB. It cleaves multiple COMPLEMENT C3 into COMPLEMENT C3A (anaphylatoxin) and COMPLEMENT C3B in the ALTERNATIVE COMPLEMENT ACTIVATION PATHWAY
  1
 

Complement 4 -- See Complement C4


A glycoprotein that is important in the activation of CLASSICAL COMPLEMENT PATHWAY. C4 is cleaved by the activated COMPLEMENT C1S into COMPLEMENT C4A and COMPLEMENT C4B
  1
 

Complement 5a -- See Complement C5a


The minor fragment formed when C5 convertase cleaves C5 into C5a and COMPLEMENT C5B. C5a is a 74-amino-acid glycopeptide with a carboxy-terminal ARGININE that is crucial for its spasmogenic activity. Of all the complement-derived anaphylatoxins, C5a is the most potent in mediating immediate hypersensitivity (HYPERSENSITIVITY, IMMEDIATE), smooth MUSCLE CONTRACTION; HISTAMINE RELEASE; and migration of LEUKOCYTES to site of INFLAMMATION
  1
 

Complement 5b -- See Complement C5b


The larger fragment generated from the cleavage of C5 by C5 CONVERTASE that yields COMPLEMENT C5A and C5b (beta chain + alpha' chain, the residual alpha chain, bound by disulfide bond). C5b remains bound to the membrane and initiates the spontaneous assembly of the late complement components to form C5b-8-poly-C9, the MEMBRANE ATTACK COMPLEX
  1
Complement Activation.   8
Complement Activation -- drug effects.   4
Complement Activation -- immunology.   4
 

Complement Activation Pathway, Alternative -- See Complement Pathway, Alternative


Complement activation initiated by the interaction of microbial ANTIGENS with COMPLEMENT C3B. When COMPLEMENT FACTOR B binds to the membrane-bound C3b, COMPLEMENT FACTOR D cleaves it to form alternative C3 CONVERTASE (C3BBB) which, stabilized by COMPLEMENT FACTOR P, is able to cleave multiple COMPLEMENT C3 to form alternative C5 CONVERTASE (C3BBB3B) leading to cleavage of COMPLEMENT C5 and the assembly of COMPLEMENT MEMBRANE ATTACK COMPLEX
  1
  Complement Activation Pathway, Classical -- 2 Related Subjects   2
Complement Activation -- physiology. : Complement and T cells / Claudia Kemper  2017 1
  Complement Activations -- 2 Related Subjects   2
 

Complement activators -- See Complement activation


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Akkadian language -- Complement. : Syntactic change in Akkadian : the evolution of sentential complementation / Guy Deutscher  2000 1
Complement C1q -- immunology. : Complement and lupus / Marina Botto  2018 1
Complement C2. : Complement in PNH and other hemolytic anaemias part 1 of 3 / Lucio Luzzatto  2017 1
Complement C3.   2
Complement C3 -- analysis. : C3 glomerulopathy / Matthew Pickering  2017 1
Complement C3 Convertase, Alternative Pathway. : Physiopathological implications of genetic variability in the complement alternative pathway / Santiago Rodríguez de Córdoba  2018 1
 

Complement C3 Convertase, CVFBb -- See Complement C3 Convertase, Alternative Pathway


A serine protease that is the complex of COMPLEMENT C3B and COMPLEMENT FACTOR BB. It cleaves multiple COMPLEMENT C3 into COMPLEMENT C3A (anaphylatoxin) and COMPLEMENT C3B in the ALTERNATIVE COMPLEMENT ACTIVATION PATHWAY
  1
Complement C3 -- immunology. : C3 glomerulopathy / Matthew Pickering  2017 1
 

Complement C3 Precursor -- See Complement C3


A glycoprotein that is central in both the classical and the alternative pathway of COMPLEMENT ACTIVATION. C3 can be cleaved into COMPLEMENT C3A and COMPLEMENT C3B, spontaneously at low level or by C3 CONVERTASE at high level. The smaller fragment C3a is an ANAPHYLATOXIN and mediator of local inflammatory process. The larger fragment C3b binds with C3 convertase to form C5 convertase
  1
Complement C3a. : Complement and organ transplantation / Steven Sacks  2018 1
Complement C4. : Complement diagnostics / Ashley Frazer-Abel  2017 1
 

Complement C4, Precursor -- See Complement C4


A glycoprotein that is important in the activation of CLASSICAL COMPLEMENT PATHWAY. C4 is cleaved by the activated COMPLEMENT C1S into COMPLEMENT C4A and COMPLEMENT C4B
  1
Complement C5a. : Complement and organ transplantation / Steven Sacks  2018 1
Complement C5b. : Complement and organ transplantation / Steven Sacks  2018 1
  Complement Complex C5b-9 -- 2 Related Subjects   2
  Complement Complex, Terminal -- 2 Related Subjects   2
 

Complement Component 2 -- See Complement C2


A component of the CLASSICAL COMPLEMENT PATHWAY. C2 is cleaved by activated COMPLEMENT C1S into COMPLEMENT C2B and COMPLEMENT C2A. C2a, the COOH-terminal fragment containing a SERINE PROTEASE, combines with COMPLEMENT C4B to form C4b2a (CLASSICAL PATHWAY C3 CONVERTASE) and subsequent C4b2a3b (CLASSICAL PATHWAY C5 CONVERTASE)
  1
 

Complement Component 3 -- See Complement C3


A glycoprotein that is central in both the classical and the alternative pathway of COMPLEMENT ACTIVATION. C3 can be cleaved into COMPLEMENT C3A and COMPLEMENT C3B, spontaneously at low level or by C3 CONVERTASE at high level. The smaller fragment C3a is an ANAPHYLATOXIN and mediator of local inflammatory process. The larger fragment C3b binds with C3 convertase to form C5 convertase
  1
 

Complement Component 3a -- See Complement C3a


The smaller fragment generated from the cleavage of complement C3 by C3 CONVERTASE. C3a, a 77-amino acid peptide, is a mediator of local inflammatory process. It induces smooth MUSCLE CONTRACTION, and HISTAMINE RELEASE from MAST CELLS and LEUKOCYTES. C3a is considered an anaphylatoxin along with COMPLEMENT C4A; COMPLEMENT C5A; and COMPLEMENT C5A, DES-ARGININE
  1
 

Complement Component 4 -- See Complement C4


A glycoprotein that is important in the activation of CLASSICAL COMPLEMENT PATHWAY. C4 is cleaved by the activated COMPLEMENT C1S into COMPLEMENT C4A and COMPLEMENT C4B
  1
 

Complement Component 5a -- See Complement C5a


The minor fragment formed when C5 convertase cleaves C5 into C5a and COMPLEMENT C5B. C5a is a 74-amino-acid glycopeptide with a carboxy-terminal ARGININE that is crucial for its spasmogenic activity. Of all the complement-derived anaphylatoxins, C5a is the most potent in mediating immediate hypersensitivity (HYPERSENSITIVITY, IMMEDIATE), smooth MUSCLE CONTRACTION; HISTAMINE RELEASE; and migration of LEUKOCYTES to site of INFLAMMATION
  1
 

Complement Component 5b -- See Complement C5b


The larger fragment generated from the cleavage of C5 by C5 CONVERTASE that yields COMPLEMENT C5A and C5b (beta chain + alpha' chain, the residual alpha chain, bound by disulfide bond). C5b remains bound to the membrane and initiates the spontaneous assembly of the late complement components to form C5b-8-poly-C9, the MEMBRANE ATTACK COMPLEX
  1
 

Complement Component, C4 -- See Complement C4


A glycoprotein that is important in the activation of CLASSICAL COMPLEMENT PATHWAY. C4 is cleaved by the activated COMPLEMENT C1S into COMPLEMENT C4A and COMPLEMENT C4B
  1
  Complement Cytolysis Inhibiting Agents -- 2 Related Subjects   2
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