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Gato (Horse) : The tale of two horses / illustrations by Caney  1949 1
 

Gator (Fish) -- See Alligator gar


  1
 

Gatorade Company -- See Gatorade (Firm)


  1
Gatorade (Firm) / http://id.loc.gov/authorities/names/n2005033632 : First in thirst : how Gatorade turned the science of sweat into a cultural phenomenon / Darren Rovell  2005 1
GATOS. : Self-assessment colour review of small animal abdominal and metabolic disorders / [edited by] Bryn Tennant  1999 1
 

Gatos (Calif.) -- See Los Gatos (Calif.)


  1
 

Gatos, Los (Calif.) -- See Los Gatos (Calif.)


  1
 

GATS (1994 April 15) -- See General Agreement on Trade in Services (1994 April 15)


  1
 

GATS (Organization) -- See General Agreement on Trade in Services (Organization)


  1
 

Gatsogiannē, Helenē -- See Gatzoyiannis, Eleni


  1
 

Gatson, Daisy Lee -- See Bates, Daisy


  1
 

GATT -- See General Agreement on Tariffs and Trade (Organization)


  1
GATT. : Regional and global regulation of international trade / edited by Francis Snyder  2002 1
 

GATT (1947 October 30) -- See General Agreement on Tariffs and Trade (1947 October 30)


  1
 

GATT International Trade Centre -- See International Trade Centre UNCTAD/GATT


  1
Gattel, Felix, -1904. : The non-authentic nature of Freud's observations / Max Scharnberg  1993 1
Gatti, Joseph Michael -- Interviews : Finis Jhung interviews Joseph Michael Gatti / by Finis Jhung & Michael Gatti Joseph  2007 1
Gattilusio, Francesco, 1332-1384 -- Family. : The Gattilusio Lordships and the Aegean World 1355-1462  2014 1
Gattinara, Mercurino, -- marchese di, -- 1465-1530 : Mercurino di Gattinara and the Creation of the Spanish Empire  2014 1
 

Gatto (1947 October 30) -- See General Agreement on Tariffs and Trade (1947 October 30)


  1
Gatto, Florence Terranova, 1939- : The scent of jasmine : vignettes from a Sicilian heritage / Florence Terranova Gatto  2007 1
Gatto, John Taylor. : Dumbing us down : the hidden curriculum of compulsory schooling / John Taylor Gatto  1992 1
Crime -- Australia -- Gatton : The Gatton mystery / James and Desmond Gibney  1977 1
Criminals -- Australia -- Gatton : The Gatton mystery / James and Desmond Gibney  1977 1
Murder -- Australia -- Gatton : The Gatton mystery / James and Desmond Gibney  1977 1
Murder -- Australia -- Queensland -- Gatton   2
Murder -- Investigation -- Australia -- Queensland -- Gatton -- Case studies : The Gatton murders : a true story of lust, vengeance and vile retribution / Stephanie Bennett  2004 1
Murder -- Australia -- Gatton(Qld.) : The Gatton mystery / James and Desmond Gibney  1977 1
Tomasi di Lampedusa, Giuseppe, 1896-1957. Gattopardo. English. : Novels for students. Volume 44 : presenting analysis, context, and criticism on commonly studied novels / Sara Constantakis, project editor ; foreword by Anne Devereaux Jordan  2014 1
Visconti, Luchino, 1906-1976. Gattopardo : Il gattopardo : Luchino Visconti / Maddalena Angelino  2004 1
Gatty, Alfred, Mrs., 1809-1873. : Mrs. Gatty and Mrs. Ewing / Christabel Maxwell  1949 1
 

Gatty, Margaret, 1809-1873 -- See Gatty, Alfred, Mrs., 1809-1873


  1
 

Gatwick, Eng -- See Gatwick (England)


  1
Gatwick (England) : Gatwick and Horley mirror (Gatwick, England)    1
 

Gatwick (Surrey) -- See Gatwick (England)


  1
 

Gatzogiannē, Helenē -- See Gatzoyiannis, Eleni


  1
Gatzoyiannis, Eleni.   2
 

Gauche divine (Group of artists) -- See Also Arts, Spanish 20th century


  1
Gaucher disease.   5
 

Gaucher Disease, Acute Neuronopathic -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Gaucher Disease, Acute Neuronopathic Type -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Gaucher Disease, Chronic -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Gaucher Disease, Chronic Neuronopathic Type -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
Gaucher Disease -- diagnosis. : Advances in Gaucher disease : basic and clinical perspectives / editor, Gregory A. Grabowski  2013 1
Gaucher Disease -- genetics. : Acid beta-Glucosidase/Glucocerebrosidase (GCase) / Gregory Grabowski  2016 1
Gaucher Disease -- immunology. : Gaucher disease : from lysosomal storage to immunopathology / Hans Aerts  2013 1
 

Gaucher Disease, Infantile -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Gaucher Disease, Infantile Cerebral -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Gaucher Disease, Juvenile -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Gaucher Disease, Juvenile and Adult, Cerebral -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
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