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Mitochondrial Diseases -- physiopathology.   2
Mitochondrial Diseases -- therapy.   2
 

Mitochondrial Disorder -- See Mitochondrial Diseases


Diseases caused by abnormal function of the MITOCHONDRIA. They may be caused by mutations, acquired or inherited, in mitochondrial DNA or in nuclear genes that code for mitochondrial components. They may also be the result of acquired mitochondria dysfunction due to adverse effects of drugs, infections, or other environmental causes
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  Mitochondrial disorders -- 2 Related Subjects   2
 

Mitochondrial DNA -- See DNA, Mitochondrial


Double-stranded DNA of MITOCHONDRIA. In eukaryotes, the mitochondrial GENOME is circular and codes for ribosomal RNAs, transfer RNAs, and about 10 proteins
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Mitochondrial DNA.   14
Mitochondrial DNA -- Abnormalities   2
Mitochondrial DNA -- Analysis.   3
 

Mitochondrial DNA disorders -- See Mitochondrial DNA Abnormalities


  1
Mitochondrial DNA -- Periodicals   3
 

Mitochondrial Dynamic -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
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Mitochondrial Dynamics.   3
 

Mitochondrial Electron Transport Chain Deficiencies -- See Mitochondrial Diseases


Diseases caused by abnormal function of the MITOCHONDRIA. They may be caused by mutations, acquired or inherited, in mitochondrial DNA or in nuclear genes that code for mitochondrial components. They may also be the result of acquired mitochondria dysfunction due to adverse effects of drugs, infections, or other environmental causes
  1
 

Mitochondrial Encephalomyopathy, Lactic Acidosis, and Stroke-Like Episodes -- See MELAS Syndrome


A mitochondrial disorder characterized by focal or generalized seizures, episodes of transient or persistent neurologic dysfunction resembling strokes, and ragged-red fibers on muscle biopsy. Affected individuals tend to be normal at birth through early childhood, then experience growth failure, episodic vomiting, and recurrent cerebral insults resulting in visual loss and hemiparesis. The cortical lesions tend to occur in the parietal and occipital lobes and are not associated with vascular occlusion. VASCULAR HEADACHE is frequently associated and the disorder tends to be familial. (From Joynt, Clinical Neurology, 1992, Ch56, p117)
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Mitochondrial Fission -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
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Mitochondrial Fission and Fusion -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
  1
 

Mitochondrial Fissions -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
  1
 

Mitochondrial Fusion -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
  1
 

Mitochondrial Fusion and Fission -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
  1
 

Mitochondrial Fusions -- See Mitochondrial Dynamics


The continuous remodeling of MITOCHONDRIA shape by fission and fusion in response to physiological conditions
  1
 

Mitochondrial Gene -- See Genes, Mitochondrial


Genes that are located on the MITOCHONDRIAL DNA. Mitochondrial inheritance is often referred to as maternal inheritance but should be differentiated from maternal inheritance that is transmitted chromosomally
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Mitochondrial Genes -- See Genes, Mitochondrial


Genes that are located on the MITOCHONDRIAL DNA. Mitochondrial inheritance is often referred to as maternal inheritance but should be differentiated from maternal inheritance that is transmitted chromosomally
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Mitochondrial genome -- See Mitochondrial DNA


  1
 

Mitochondrial genomes -- See Mitochondrial DNA


  1
 

Mitochondrial Inheritance -- See Genes, Mitochondrial


Genes that are located on the MITOCHONDRIAL DNA. Mitochondrial inheritance is often referred to as maternal inheritance but should be differentiated from maternal inheritance that is transmitted chromosomally
  1
 

Mitochondrial Membrane -- See Mitochondrial Membranes


The two lipoprotein layers in the MITOCHONDRION. The outer membrane encloses the entire mitochondrion and contains channels with TRANSPORT PROTEINS to move molecules and ions in and out of the organelle. The inner membrane folds into cristae and contains many ENZYMES important to cell METABOLISM and energy production (MITOCHONDRIAL ATP SYNTHASE)
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Mitochondrial Membrane, Inner -- See Mitochondrial Membranes


The two lipoprotein layers in the MITOCHONDRION. The outer membrane encloses the entire mitochondrion and contains channels with TRANSPORT PROTEINS to move molecules and ions in and out of the organelle. The inner membrane folds into cristae and contains many ENZYMES important to cell METABOLISM and energy production (MITOCHONDRIAL ATP SYNTHASE)
  1
 

Mitochondrial Membrane, Outer -- See Mitochondrial Membranes


The two lipoprotein layers in the MITOCHONDRION. The outer membrane encloses the entire mitochondrion and contains channels with TRANSPORT PROTEINS to move molecules and ions in and out of the organelle. The inner membrane folds into cristae and contains many ENZYMES important to cell METABOLISM and energy production (MITOCHONDRIAL ATP SYNTHASE)
  1
Mitochondrial Membrane Transport Proteins.   3
Mitochondrial membranes.   5
 

Mitochondrial Membranes, Inner -- See Mitochondrial Membranes


The two lipoprotein layers in the MITOCHONDRION. The outer membrane encloses the entire mitochondrion and contains channels with TRANSPORT PROTEINS to move molecules and ions in and out of the organelle. The inner membrane folds into cristae and contains many ENZYMES important to cell METABOLISM and energy production (MITOCHONDRIAL ATP SYNTHASE)
  1
Mitochondrial Membranes -- metabolism. : Initiation and elongation of mammalian mitochondrial protein synthesis / Linda L. Spremulli  2018 1
 

Mitochondrial Membranes, Outer -- See Mitochondrial Membranes


The two lipoprotein layers in the MITOCHONDRION. The outer membrane encloses the entire mitochondrion and contains channels with TRANSPORT PROTEINS to move molecules and ions in and out of the organelle. The inner membrane folds into cristae and contains many ENZYMES important to cell METABOLISM and energy production (MITOCHONDRIAL ATP SYNTHASE)
  1
 

Mitochondrial Myopathy, Encephalopathy, Lactic Acidosis, And Stroke-Like Episodes -- See MELAS Syndrome


A mitochondrial disorder characterized by focal or generalized seizures, episodes of transient or persistent neurologic dysfunction resembling strokes, and ragged-red fibers on muscle biopsy. Affected individuals tend to be normal at birth through early childhood, then experience growth failure, episodic vomiting, and recurrent cerebral insults resulting in visual loss and hemiparesis. The cortical lesions tend to occur in the parietal and occipital lobes and are not associated with vascular occlusion. VASCULAR HEADACHE is frequently associated and the disorder tends to be familial. (From Joynt, Clinical Neurology, 1992, Ch56, p117)
  1
 

Mitochondrial Myopathy, Lactic Acidosis, Stroke-Like Episode -- See MELAS Syndrome


A mitochondrial disorder characterized by focal or generalized seizures, episodes of transient or persistent neurologic dysfunction resembling strokes, and ragged-red fibers on muscle biopsy. Affected individuals tend to be normal at birth through early childhood, then experience growth failure, episodic vomiting, and recurrent cerebral insults resulting in visual loss and hemiparesis. The cortical lesions tend to occur in the parietal and occipital lobes and are not associated with vascular occlusion. VASCULAR HEADACHE is frequently associated and the disorder tends to be familial. (From Joynt, Clinical Neurology, 1992, Ch56, p117)
  1
 

Mitochondrial pathology -- See Also the narrower term Mitochondrial DNA Abnormalities


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Mitochondrial pathology.   41
Mitochondrial pathology -- Case studies. : Mitochondrial case studies : underlying mechanisms and diagnosis / edited by Russell P. Saneto, Department of Neurology/Division of Pediatric Neurology, Seattle Children's Hospital/University of Washington, Seattle, WA, USA, Sumit Parikh, Cleveland Clinic Lerner College of Medicine & Case Western Reserve University, Cleveland, OH, USA ; Neurogenetics, Metabolism and Mitochondrial Disease Center, Cleveland Clinic, Cleveland, OH, USA, Bruce H. Cohen, Northeast Ohio Medical University, Rootstown, OH, USA ; The NeuroDevelopmental Science Center and Division of Neurology, Department of Pediatrics, Children's Hospital and Medical Center of Akron, Akron, OH, USA  2016 1
Mitochondrial pathology -- Diagnosis -- Case studies. : Mitochondrial case studies : underlying mechanisms and diagnosis / edited by Russell P. Saneto, Department of Neurology/Division of Pediatric Neurology, Seattle Children's Hospital/University of Washington, Seattle, WA, USA, Sumit Parikh, Cleveland Clinic Lerner College of Medicine & Case Western Reserve University, Cleveland, OH, USA ; Neurogenetics, Metabolism and Mitochondrial Disease Center, Cleveland Clinic, Cleveland, OH, USA, Bruce H. Cohen, Northeast Ohio Medical University, Rootstown, OH, USA ; The NeuroDevelopmental Science Center and Division of Neurology, Department of Pediatrics, Children's Hospital and Medical Center of Akron, Akron, OH, USA  2016 1
Mitochondrial pathology -- Handbooks, manuals, etc : Handbook of mitochondrial dysfunction / edited by Shamim I. Ahmad  2019 1
mitochondrial permeability transition pore : Mitochondria and calcium signaling in cell life and cell death / Michael R. Duchen  2018 1
 

Mitochondrial Protein -- See Mitochondrial Proteins


Proteins encoded by the mitochondrial genome or proteins encoded by the nuclear genome that are imported to and resident in the MITOCHONDRIA
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Mitochondrial Proteins.   6
Mitochondrial Proteins -- biosynthesis. : Initiation and elongation of mammalian mitochondrial protein synthesis / Linda L. Spremulli  2018 1
Mitochondrial Proteins -- chemistry.   2
Mitochondrial Proteins -- metabolism.   6
 

Mitochondrial Respiratory Chain Deficiencies -- See Mitochondrial Diseases


Diseases caused by abnormal function of the MITOCHONDRIA. They may be caused by mutations, acquired or inherited, in mitochondrial DNA or in nuclear genes that code for mitochondrial components. They may also be the result of acquired mitochondria dysfunction due to adverse effects of drugs, infections, or other environmental causes
  1
 

Mitochondrion -- See Mitochondria


Semiautonomous, self-reproducing organelles that occur in the cytoplasm of all cells of most, but not all, eukaryotes. Each mitochondrion is surrounded by a double limiting membrane. The inner membrane is highly invaginated, and its projections are called cristae. Mitochondria are the sites of the reactions of oxidative phosphorylation, which result in the formation of ATP. They contain distinctive RIBOSOMES, transfer RNAs (RNA, TRANSFER); AMINO ACYL T RNA SYNTHETASES; and elongation and termination factors. Mitochondria depend upon genes within the nucleus of the cells in which they reside for many essential messenger RNAs (RNA, MESSENGER). Mitochondria are believed to have arisen from aerobic bacteria that established a symbiotic relationship with primitive protoeukaryotes. (King & Stansfield, A Dictionary of Genetics, 4th ed)
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Mitochondrion, Heart -- See Mitochondria, Heart


The mitochondria of the myocardium
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Mitochondrion, Muscle -- See Mitochondria, Muscle


Mitochondria of skeletal and smooth muscle. It does not include myocardial mitochondria for which MITOCHONDRIA, HEART is available
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