Watson, Richard, 1781-1833 -- Influence : The role of Richard Watson's Theological institutes in the development of Methodism after John Wesley / Barry W. Hamilton ; with a foreword by Jason E. Vickers
2014
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Watson, Richard, 1781-1833. Theological institutes : The role of Richard Watson's Theological institutes in the development of Methodism after John Wesley / Barry W. Hamilton ; with a foreword by Jason E. Vickers
Watson, Sam, 1952- -- Interviews : Aboriginal authors or themes. 2 : interviews with the following authors: Sally Morgan, Bill Rosser, Mudrooroo Narogin, Roberta Sykes, Sam Watson / produced by SBS
2012
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Watson, Samuel, 1785-1818 -- Trials, litigation, etc : Trials by court martial of Capt. Samuel Watson, 2d, David Livermore, Daniel Kent, and William Prouty of the 1st Reg. 1st Brig. and 7th Division of Massachusetts Militia, 1810
An autosomal dominant inherited disorder (with a high frequency of spontaneous mutations) that features developmental changes in the nervous system, muscles, bones, and skin, most notably in tissue derived from the embryonic NEURAL CREST. Multiple hyperpigmented skin lesions and subcutaneous tumors are the hallmark of this disease. Peripheral and central nervous system neoplasms occur frequently, especially OPTIC NERVE GLIOMA and NEUROFIBROSARCOMA. NF1 is caused by mutations which inactivate the NF1 gene (GENES, NEUROFIBROMATOSIS 1) on chromosome 17q. The incidence of learning disabilities is also elevated in this condition. (From Adams et al., Principles of Neurology, 6th ed, pp1014-18) There is overlap of clinical features with NOONAN SYNDROME in a syndrome called neurofibromatosis-Noonan syndrome. Both the PTPN11 and NF1 gene products are involved in the SIGNAL TRANSDUCTION pathway of Ras (RAS PROTEINS)
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Watson, Thomas David : Investigation into the death of Ian William Turner : Coroner's findings / [Hal Hallenstein, State Coroner, Victoria]
