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Mark   Year Entries
Autoimmune diseases in pregnancy. : HIV in pregnancy and childbirth  2003 1
Autoimmune diseases in pregnancy -- Complications -- Handbooks, manuals, etc : Reproductive and hormonal aspects of systemic autoimmune diseases / edited by Michael Lockshin and D. Ware Branch  2006 1
Autoimmune diseases in women. : Women's health in autoimmune diseases / Shefali Khanna Sharma, editor  2020 1
Autoimmune diseases in women -- Patients -- Employment : Women, work, and autoimmune disease : keep working, girlfriend! / Rosalind Joffe and Joan Friedlander ; edited by L.G. Mansfield  2008 1
Autoimmune Diseases -- Infant, Newborn. : Neonatal immunity / by Constantin Bona ; foreword by Noel R. Rose  2005 1
Autoimmune diseases -- Laboratory manuals   2
Autoimmune Diseases [MESH] : Culture, society and sexuality : a reader / edited by Richard Parker and Peter Aggleton  1999 1
Autoimmune diseases -- Microbiology   2
Autoimmune diseases -- Molecular aspects   2
Autoimmune Diseases of the Nervous System   2
Autoimmune Diseases of the Nervous System -- diagnosis   2
Autoimmune Diseases of the Nervous System -- etiology : Neural and neuroendocrine mechanisms in host defense and autoimmunity / edited by C. Jane Welsh, Mary W. Meagher, Esther M. Sternberg  2006 1
Autoimmune Diseases of the Nervous System -- immunology   3
Autoimmune Diseases of the Nervous System -- physiopathology. : Neuroimmunology in clinical practice / edited by Bernadette Kalman and Thomas H. Brannagan III  2008 1
Autoimmune Diseases of the Nervous System -- therapy   2
Autoimmune diseases -- Pathogenesis   2
Autoimmune Diseases -- pathology   2
Autoimmune diseases -- Pathophysiology : Neuroinflammation (David)  2014 1
Autoimmune diseases -- Periodicals   10
Autoimmune Diseases -- physiopathology   10
Autoimmune diseases -- Poetry : Hinge / Molly Spencer  2020 1
Autoimmune diseases -- Psychology : Chronic illness, vulnerability and social work : autoimmunity and the contemporary disease experience / written by Liz Price and Liz Walker  2015 1
Autoimmune diseases -- Research : Passion for excellence : my lifelong journey into medicine and public service / Haralampos M. Moutsopoulos  2022 1
Autoimmune diseases -- Surgery : Surgery in rheumatic and musculoskeletal disease / editors, Fabiola Atzeni, Ignazio Francesco Masala, Daniel Aletaha, Marcus Lee, Xenofon Baraliakos  2018 1
Autoimmune diseases -- Therapeutic use : Biologic and gene therapy of autoimmune disease / volume editor, C.G. Fathman  2000 1
Autoimmune Diseases -- therapy   16
Autoimmune diseases -- Treatment.   9
 

Autoimmune hemolytic anemia -- See Hemolytic anemia, Autoimmune


  1
 

Autoimmune hemolytic disorder -- See Hemolytic anemia, Autoimmune


  1
 

Autoimmune Hepatitides -- See Hepatitis, Autoimmune


A chronic self-perpetuating hepatocellular INFLAMMATION of unknown cause, usually with HYPERGAMMAGLOBULINEMIA and serum AUTOANTIBODIES
  1
  Autoimmune hepatitis -- 2 Related Subjects   2
 

Autoimmune Lymphoproliferative Syndrome -- See Also fas Receptor


A tumor necrosis factor receptor subtype found in a variety of tissues and on activated LYMPHOCYTES. It has specificity for FAS LIGAND and plays a role in regulation of peripheral immune responses and APOPTOSIS. Multiple isoforms of the protein exist due to multiple ALTERNATIVE SPLICING. The activated receptor signals via a conserved death domain that associates with specific TNF RECEPTOR-ASSOCIATED FACTORS in the CYTOPLASM. Mutations in the CD95 gene are associated with cases of autoimmune lymphoproliferative syndrome
  1
Autoimmune Lymphoproliferative Syndrome : Apoptosis and its relevance to autoimmunity / volume editor, Keith B. Elkon  2006 1
 

Autoimmune Lymphoproliferative Syndrome Type 1, Autosomal Dominant -- See Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Autoimmune Lymphoproliferative Syndrome Type 2B -- See Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Autoimmune Lymphoproliferative Syndrome Type 2B (ALPS2B) -- See Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Autoimmune Lymphoproliferative Syndrome, Type I, Autosomal Dominant -- See Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Autoimmune Lymphoproliferative Syndrome, Type IIb -- See Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Autoimmune Lymphoproliferative Syndromes -- See Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Autoimmune Neuropathies, Acute -- See Guillain-Barre Syndrome


An acute inflammatory autoimmune neuritis caused by T cell- mediated cellular immune response directed towards peripheral myelin. Demyelination occurs in peripheral nerves and nerve roots. The process is often preceded by a viral or bacterial infection, surgery, immunization, lymphoma, or exposure to toxins. Common clinical manifestations include progressive weakness, loss of sensation, and loss of deep tendon reflexes. Weakness of respiratory muscles and autonomic dysfunction may occur. (From Adams et al., Principles of Neurology, 6th ed, pp1312-1314)
  1
 

Autoimmune Neuropathy, Acute -- See Guillain-Barre Syndrome


An acute inflammatory autoimmune neuritis caused by T cell- mediated cellular immune response directed towards peripheral myelin. Demyelination occurs in peripheral nerves and nerve roots. The process is often preceded by a viral or bacterial infection, surgery, immunization, lymphoma, or exposure to toxins. Common clinical manifestations include progressive weakness, loss of sensation, and loss of deep tendon reflexes. Weakness of respiratory muscles and autonomic dysfunction may occur. (From Adams et al., Principles of Neurology, 6th ed, pp1312-1314)
  1
 

Autoimmune Response -- See Autoimmunity


Process whereby the immune system reacts against the body's own tissues. Autoimmunity may produce or be caused by AUTOIMMUNE DISEASES
  1
 

Autoimmune Responses -- See Autoimmunity


Process whereby the immune system reacts against the body's own tissues. Autoimmunity may produce or be caused by AUTOIMMUNE DISEASES
  1
 

Autoimmune Thrombocytopenia -- See Purpura, Thrombocytopenic, Idiopathic


Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
  1
 

Autoimmune Thrombocytopenias -- See Purpura, Thrombocytopenic, Idiopathic


Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
  1
 

Autoimmune Thrombocytopenic Purpura -- See Purpura, Thrombocytopenic, Idiopathic


Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
  1
 

Autoimmune Thrombocytopenic Purpuras -- See Purpura, Thrombocytopenic, Idiopathic


Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
  1
Autoimmune thyroiditis. : Undoing Hashimoto's : a Guide to Managing Symptoms, Relieving Overwhelm & Living Well  2018 1
Autoimmunit├Ąt   2
Apoptosis -- Autoimmunit├Ąt -- Aufsatzsammlung. : Apoptosis and its relevance to autoimmunity / volume editor, Keith B. Elkon  2006 1
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