Kaller, Maximilian, 1880-1947 -- Congresses : Maximilian Kaller, Bischof der wandernden Kirche : Flucht und Vertreibung--Integration--Brückenbau / herausgegeben von Thomas Flammer und Hans-Jürgen Karp
Kallikrein -- Genetics : Kallikrein-related peptidases. Volume 1, Characterization, regulation, and interactions within the protease web / Viktor Magdolen [and others], editors
A glycoprotein that is a kallikrein-like serine proteinase and an esterase, produced by epithelial cells of both normal and malignant prostate tissue. It is an important marker for the diagnosis of prostate cancer
A BLOOD PRESSURE regulating system of interacting components that include RENIN; ANGIOTENSINOGEN; ANGIOTENSIN CONVERTING ENZYME; ANGIOTENSIN I; ANGIOTENSIN II; and angiotensinase. Renin, an enzyme produced in the kidney, acts on angiotensinogen, an alpha-2 globulin produced by the liver, forming ANGIOTENSIN I. Angiotensin-converting enzyme, contained in the lung, acts on angiotensin I in the plasma converting it to ANGIOTENSIN II, an extremely powerful vasoconstrictor. Angiotensin II causes contraction of the arteriolar and renal VASCULAR SMOOTH MUSCLE, leading to retention of salt and water in the KIDNEY and increased arterial blood pressure. In addition, angiotensin II stimulates the release of ALDOSTERONE from the ADRENAL CORTEX, which in turn also increases salt and water retention in the kidney. Angiotensin-converting enzyme also breaks down BRADYKININ, a powerful vasodilator and component of the KALLIKREIN-KININ SYSTEM
Kallikrein -- Metabolism : Kallikrein-related peptidases. Volume 1, Characterization, regulation, and interactions within the protease web / Viktor Magdolen [and others], editors
Kallir, Otto, 1894-1978. : Gustav Klimt--Egon Schiele : in commemoration of the achievements of Dr. Otto Kallir / Jane Kallir ; forewords by Thomas M. Messer and Allessandra Comini
Kallmann, McKinnell & Knowles. / http://id.loc.gov/authorities/names/no2016090532 : Concrete changes : architecture, politics, and the design of Boston City Hall / Brian M. Sirman
2018
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Kallmann, McKinnell & Wood -- Exhibitions. / http://id.loc.gov/authorities/names/nr88009956 http://id.loc.gov/authorities/subjects/sh99001275 : The Architecture of Kallmann, McKinnell & Wood / edited by Alex Krieger
1988
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Kallmann Syndrome : Abnormalities in puberty : scientific and clinical advances / volume editor, Henriette Delemarre-Van de Waal
A genetically heterogeneous disorder caused by hypothalamic GNRH deficiency and OLFACTORY NERVE defects. It is characterized by congenital HYPOGONADOTROPIC HYPOGONADISM and ANOSMIA, possibly with additional midline defects. It can be transmitted as an X-linked (GENETIC DISEASES, X-LINKED), an autosomal dominant, or an autosomal recessive trait
A genetically heterogeneous disorder caused by hypothalamic GNRH deficiency and OLFACTORY NERVE defects. It is characterized by congenital HYPOGONADOTROPIC HYPOGONADISM and ANOSMIA, possibly with additional midline defects. It can be transmitted as an X-linked (GENETIC DISEASES, X-LINKED), an autosomal dominant, or an autosomal recessive trait
A genetically heterogeneous disorder caused by hypothalamic GNRH deficiency and OLFACTORY NERVE defects. It is characterized by congenital HYPOGONADOTROPIC HYPOGONADISM and ANOSMIA, possibly with additional midline defects. It can be transmitted as an X-linked (GENETIC DISEASES, X-LINKED), an autosomal dominant, or an autosomal recessive trait
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Kallmann Syndrome -- genetics : Kallmann syndrome and hypogonadotropic hypogonadism / volume editor, Richard Quinton
A genetically heterogeneous disorder caused by hypothalamic GNRH deficiency and OLFACTORY NERVE defects. It is characterized by congenital HYPOGONADOTROPIC HYPOGONADISM and ANOSMIA, possibly with additional midline defects. It can be transmitted as an X-linked (GENETIC DISEASES, X-LINKED), an autosomal dominant, or an autosomal recessive trait
A genetically heterogeneous disorder caused by hypothalamic GNRH deficiency and OLFACTORY NERVE defects. It is characterized by congenital HYPOGONADOTROPIC HYPOGONADISM and ANOSMIA, possibly with additional midline defects. It can be transmitted as an X-linked (GENETIC DISEASES, X-LINKED), an autosomal dominant, or an autosomal recessive trait
A genetically heterogeneous disorder caused by hypothalamic GNRH deficiency and OLFACTORY NERVE defects. It is characterized by congenital HYPOGONADOTROPIC HYPOGONADISM and ANOSMIA, possibly with additional midline defects. It can be transmitted as an X-linked (GENETIC DISEASES, X-LINKED), an autosomal dominant, or an autosomal recessive trait
