Keramischer Supraleiter : Corrosion of glass, ceramics, and ceramic superconductors : principles, testing, characterization and applications / edited by David E. Clark and Bruce K. Zoitos
Kerang Court records Australia Victoria : Register of convictions, orders and other proceedings in the court of Petty Sessions at Kerang, commencing 2/5/1888 to 28/1/1920 January 1983 1888-1920
Kerang Lakes Region (Vic.) : The Kerang Lakes area draft management plan / prepared by the Kerang Lakes Area Working Group
1992
1
Kerang Lakes Salinity Australia Victoria : Further considerations of the environmental effects of salinity on waterbodies in the Kerang Lakes management area / John McGuckin
Kerang Region Registers of births, etc Australia Victoria : Home papers please copy : births, marriages, deaths, obituaries (and other relevant information) from the pages of the Kerang Times & Swan Hill Gazette ; circulating in Kerang, Swan Hill, Cohuna, Durham Ox, Terricks, Boort, Gunbower and surrounding districts / compiled by Jenny Bottcher and Jean Hosking
Kerang Region (Vic.) -- Genealogy : Home papers please copy : births, marriages, deaths, obituaries (and other relevant information) from the pages of the Kerang Times & Swan Hill Gazette ; circulating in Kerang, Swan Hill, Cohuna, Durham Ox, Terricks, Boort, Gunbower and surrounding districts / compiled by Jenny Bottcher and Jean Hosking
An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
