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Myelodysplastic syndromes -- Handbooks, manuals, etc : Clinician's manual on myelodysplastic syndromes / Alan List  2008 1
Myelodysplastic syndromes -- Pathophysiology : Novel insights into pathophysiology, diagnostics and treatment of myelodysplastic syndromes / editors, Rainer Haas, Ulrich Germing  2014 1
Myelodysplastic Syndromes -- physiopathology   2
Myelodysplastic syndromes -- Research : Myelodysplastic syndromes (MDS) : risk factors, treatment and prognosis / Deanna Rodgers, editor  2016 1
Myelodysplastic Syndromes -- therapy   4
Myelodysplastic syndromes -- Treatment   3
Myelodysplastic syndromes -- Treatment -- Popular works. : Understanding myelodysplastic syndromes (MDS) : a guide for patients and families / Leukaemia Foundation  2006 1
 

Myeloencephalitides -- See Encephalomyelitis


A general term indicating inflammation of the BRAIN and SPINAL CORD, often used to indicate an infectious process, but also applicable to a variety of autoimmune and toxic-metabolic conditions. There is significant overlap regarding the usage of this term and ENCEPHALITIS in the literature
  1
 

Myeloencephalitis -- See Encephalomyelitis


A general term indicating inflammation of the BRAIN and SPINAL CORD, often used to indicate an infectious process, but also applicable to a variety of autoimmune and toxic-metabolic conditions. There is significant overlap regarding the usage of this term and ENCEPHALITIS in the literature
  1
Myelogenese : Magnetic resonance of myelination and myelin disorders / Marjo S. van der Knaap, Jaap Valk ; with contributions by F. Barkhof [and others]  2005 1
  Myelogenous Leukemia -- 2 Related Subjects   2
 

Myelogenous Leukemia, Acute -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myelogenous Leukemia, Chronic -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myelogenous Leukemia, Ph1-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myelogenous Leukemias -- See Leukemia, Myeloid


Form of leukemia characterized by an uncontrolled proliferation of the myeloid lineage and their precursors (MYELOID PROGENITOR CELLS) in the bone marrow and other sites
  1
 

Myelogenous Leukemias, Acute -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myelogenous Leukemias, Chronic -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myelogenous Leukemias, Ph1-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
Myelography. : Myelopathy : a guide to diagnosis and treatment / Benjamin Greenberg, editor  2022 1
 

Myeloid Cell -- See Myeloid Cells


The classes of BONE MARROW-derived blood cells in the monocytic series (MONOCYTES and their precursors) and granulocytic series (GRANULOCYTES and their precursors)
  1
 

Myeloid Cell-Growth Inducer -- See Colony-Stimulating Factors


Glycoproteins found in a subfraction of normal mammalian plasma and urine. They stimulate the proliferation of bone marrow cells in agar cultures and the formation of colonies of granulocytes and/or macrophages. The factors include INTERLEUKIN-3; (IL-3); GRANULOCYTE COLONY-STIMULATING FACTOR; (G-CSF); MACROPHAGE COLONY-STIMULATING FACTOR; (M-CSF); and GRANULOCYTE-MACROPHAGE COLONY-STIMULATING FACTOR; (GM-CSF)
  1
Myeloid Cells : Neutrophil methods and protocols / edited by Mark T. Quinn, Frank R. DeLeo  2014 1
Myeloid-Derived Suppressor Cells : Myeloid-derived suppressor cells / edited by Sven Brandau, Anca Dorhoi  2021 1
Myeloid-Derived Suppressor Cells -- pathology : Tumor microenvironment : hematopoietic cells. Part A / Alexander Birbrair, editor  2020 1
  Myeloid differentiation-inducing protein -- 2 Related Subjects   2
 

Myeloid growth inducer-2 -- See Interleukin-6


  1
  Myeloid Leukemia -- 3 Related Subjects   3
Myeloid leukemia   2
 

Myeloid Leukemia, Acute -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myeloid Leukemia, Acute, M1 -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myeloid Leukemia, Acute, M2 -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
Myeloid leukemia -- Atlases : Myeloid malignancies : an atlas of investigation and diagnosis / Barbara J. Bain, Estella Matutes  2010 1
 

Myeloid Leukemia, Chronic -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
Myeloid leukemia -- Patients -- United States -- Biography. : Time on fire : my comedy of terrors / by Evan Handler  1997 1
 

Myeloid Leukemia, Ph1-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemia, Philadelphia-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemias -- See Leukemia, Myeloid


Form of leukemia characterized by an uncontrolled proliferation of the myeloid lineage and their precursors (MYELOID PROGENITOR CELLS) in the bone marrow and other sites
  1
 

Myeloid Leukemias, Acute -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myeloid Leukemias, Chronic -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemias, Ph1-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemias, Philadelphia-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloma, Multiple -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myeloma-Multiples -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myeloma, Plasma-Cell -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
  Myeloma proteins -- 2 Related Subjects   2
Myeloma proteins -- Analysis : Multiple myeloma and related serum protein disorders : an electrophoretic guide / Neil S. Harris, William E. Winter  2012 1
 

Myeloma, Smoldering -- See Smoldering Multiple Myeloma


An asymptomatic and slow-growing PLASMA CELL dyscrasia characterized by presence of MYELOMA PROTEINS and clonal bone marrow plasma cells without end-organ damage (e.g., renal impairment). It is distinguished from MONOCLONAL GAMMOPATHY OF UNDETERMINED SIGNIFICANCE by a much higher risk of progression to symptomatic MULTIPLE MYELOMA
  1
 

Myeloma-spleen cell hybrids -- See Hybridomas


  1
 

Myelomas, Multiple -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myelomas, Plasma Cell -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
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