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Myelography. : Myelopathy : a guide to diagnosis and treatment / Benjamin Greenberg, editor  2022 1
 

Myeloid Cell -- See Myeloid Cells


The classes of BONE MARROW-derived blood cells in the monocytic series (MONOCYTES and their precursors) and granulocytic series (GRANULOCYTES and their precursors)
  1
 

Myeloid Cell-Growth Inducer -- See Colony-Stimulating Factors


Glycoproteins found in a subfraction of normal mammalian plasma and urine. They stimulate the proliferation of bone marrow cells in agar cultures and the formation of colonies of granulocytes and/or macrophages. The factors include INTERLEUKIN-3; (IL-3); GRANULOCYTE COLONY-STIMULATING FACTOR; (G-CSF); MACROPHAGE COLONY-STIMULATING FACTOR; (M-CSF); and GRANULOCYTE-MACROPHAGE COLONY-STIMULATING FACTOR; (GM-CSF)
  1
Myeloid Cells : Neutrophil methods and protocols / edited by Mark T. Quinn, Frank R. DeLeo  2014 1
Myeloid-Derived Suppressor Cells : Myeloid-derived suppressor cells / edited by Sven Brandau, Anca Dorhoi  2021 1
Myeloid-Derived Suppressor Cells -- pathology : Tumor microenvironment : hematopoietic cells. Part A / Alexander Birbrair, editor  2020 1
  Myeloid differentiation-inducing protein -- 2 Related Subjects   2
 

Myeloid growth inducer-2 -- See Interleukin-6


  1
  Myeloid Leukemia -- 3 Related Subjects   3
Myeloid leukemia   2
 

Myeloid Leukemia, Acute -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myeloid Leukemia, Acute, M1 -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myeloid Leukemia, Acute, M2 -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
Myeloid leukemia -- Atlases : Myeloid malignancies : an atlas of investigation and diagnosis / Barbara J. Bain, Estella Matutes  2010 1
 

Myeloid Leukemia, Chronic -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
Myeloid leukemia -- Patients -- United States -- Biography. : Time on fire : my comedy of terrors / by Evan Handler  1997 1
 

Myeloid Leukemia, Ph1-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemia, Philadelphia-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemias -- See Leukemia, Myeloid


Form of leukemia characterized by an uncontrolled proliferation of the myeloid lineage and their precursors (MYELOID PROGENITOR CELLS) in the bone marrow and other sites
  1
 

Myeloid Leukemias, Acute -- See Leukemia, Myeloid, Acute


Clonal expansion of myeloid blasts in bone marrow, blood, and other tissue. Myeloid leukemias develop from changes in cells that normally produce NEUTROPHILS; BASOPHILS; EOSINOPHILS; and MONOCYTES
  1
 

Myeloid Leukemias, Chronic -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemias, Ph1-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloid Leukemias, Philadelphia-Positive -- See Leukemia, Myelogenous, Chronic, BCR-ABL Positive


Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS
  1
 

Myeloma, Multiple -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myeloma-Multiples -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myeloma, Plasma-Cell -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
  Myeloma proteins -- 2 Related Subjects   2
Myeloma proteins -- Analysis : Multiple myeloma and related serum protein disorders : an electrophoretic guide / Neil S. Harris, William E. Winter  2012 1
 

Myeloma, Smoldering -- See Smoldering Multiple Myeloma


An asymptomatic and slow-growing PLASMA CELL dyscrasia characterized by presence of MYELOMA PROTEINS and clonal bone marrow plasma cells without end-organ damage (e.g., renal impairment). It is distinguished from MONOCLONAL GAMMOPATHY OF UNDETERMINED SIGNIFICANCE by a much higher risk of progression to symptomatic MULTIPLE MYELOMA
  1
 

Myeloma-spleen cell hybrids -- See Hybridomas


  1
 

Myelomas, Multiple -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myelomas, Plasma Cell -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myelomatoses -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myelomatosis -- See Multiple Myeloma


A malignancy of mature PLASMA CELLS engaging in monoclonal immunoglobulin production. It is characterized by hyperglobulinemia, excess Bence-Jones proteins (free monoclonal IMMUNOGLOBULIN LIGHT CHAINS) in the urine, skeletal destruction, bone pain, and fractures. Other features include ANEMIA; HYPERCALCEMIA; and RENAL INSUFFICIENCY
  1
 

Myelomeningocele -- See Meningomyelocele


Congenital, or rarely acquired, herniation of meningeal and spinal cord tissue through a bony defect in the vertebral column. The majority of these defects occur in the lumbosacral region. Clinical features include PARAPLEGIA, loss of sensation in the lower body, and incontinence. This condition may be associated with the ARNOLD-CHIARI MALFORMATION and HYDROCEPHALUS. (From Joynt, Clinical Neurology, 1992, Ch55, pp35-6)
  1
Myelomeningocele.   2
 

Myelomeningocele, Acquired -- See Meningomyelocele


Congenital, or rarely acquired, herniation of meningeal and spinal cord tissue through a bony defect in the vertebral column. The majority of these defects occur in the lumbosacral region. Clinical features include PARAPLEGIA, loss of sensation in the lower body, and incontinence. This condition may be associated with the ARNOLD-CHIARI MALFORMATION and HYDROCEPHALUS. (From Joynt, Clinical Neurology, 1992, Ch55, pp35-6)
  1
Myelomeningocele -- Congresses. : Spina bifida : a multidisciplinary approach / Robert L. McLaurin, chief editor ; Sonya Oppenheimer, Luciano Dias, and William E. Kaplan, co-editors  1986 1
Myelomeningocele -- Treatment : The treatment of the baby with myelomeningocele to enhance lower extremity function / by Barbara J. Bour  1995 1
 

Myelomeningoceles -- See Meningomyelocele


Congenital, or rarely acquired, herniation of meningeal and spinal cord tissue through a bony defect in the vertebral column. The majority of these defects occur in the lumbosacral region. Clinical features include PARAPLEGIA, loss of sensation in the lower body, and incontinence. This condition may be associated with the ARNOLD-CHIARI MALFORMATION and HYDROCEPHALUS. (From Joynt, Clinical Neurology, 1992, Ch55, pp35-6)
  1
 

Myelomeningoceles, Acquired -- See Meningomyelocele


Congenital, or rarely acquired, herniation of meningeal and spinal cord tissue through a bony defect in the vertebral column. The majority of these defects occur in the lumbosacral region. Clinical features include PARAPLEGIA, loss of sensation in the lower body, and incontinence. This condition may be associated with the ARNOLD-CHIARI MALFORMATION and HYDROCEPHALUS. (From Joynt, Clinical Neurology, 1992, Ch55, pp35-6)
  1
 

Myelon -- See Spinal Cord


A cylindrical column of tissue that lies within the vertebral canal. It is composed of WHITE MATTER and GRAY MATTER

--consider also terms at MYEL-
  1
 

Myelons -- See Spinal Cord


A cylindrical column of tissue that lies within the vertebral canal. It is composed of WHITE MATTER and GRAY MATTER

--consider also terms at MYEL-
  1
 

Myelopathic muscular atrophy -- See Spinal muscular atrophy


  1
 

Myelopathies -- See Spinal Cord Diseases


Pathologic conditions which feature SPINAL CORD damage or dysfunction, including disorders involving the meninges and perimeningeal spaces surrounding the spinal cord. Traumatic injuries, vascular diseases, infections, and inflammatory/autoimmune processes may affect the spinal cord
  1
 

Myelopathies, Ischemic -- See Spinal Cord Ischemia


Reduced blood flow to the spinal cord which is supplied by the anterior spinal artery and the paired posterior spinal arteries. This condition may be associated with ARTERIOSCLEROSIS, trauma, emboli, diseases of the aorta, and other disorders. Prolonged ischemia may lead to INFARCTION of spinal cord tissue
  1
 

Myelopathies, Post-Traumatic -- See Spinal Cord Injuries


Penetrating and non-penetrating injuries to the spinal cord resulting from traumatic external forces (e.g., WOUNDS, GUNSHOT; WHIPLASH INJURIES; etc.)
  1
 

Myelopathies, Traumatic -- See Spinal Cord Injuries


Penetrating and non-penetrating injuries to the spinal cord resulting from traumatic external forces (e.g., WOUNDS, GUNSHOT; WHIPLASH INJURIES; etc.)
  1
 

Myelopathy -- See Spinal Cord Diseases


Pathologic conditions which feature SPINAL CORD damage or dysfunction, including disorders involving the meninges and perimeningeal spaces surrounding the spinal cord. Traumatic injuries, vascular diseases, infections, and inflammatory/autoimmune processes may affect the spinal cord
  1
 

Myelopathy, Ischemic -- See Spinal Cord Ischemia


Reduced blood flow to the spinal cord which is supplied by the anterior spinal artery and the paired posterior spinal arteries. This condition may be associated with ARTERIOSCLEROSIS, trauma, emboli, diseases of the aorta, and other disorders. Prolonged ischemia may lead to INFARCTION of spinal cord tissue
  1
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