Prion diseases -- Diagnosis -- United States : Advancing prion science : guidance for the National Prion Research Program, interim report / Rick Erdtmann and Laura Sivitz, editors ; Committee on Transmissible Spongiform Encephalopathies: Assessment of Relevant Science, Medical Follow-Up Agency, Institute of Medicine of the National Academies
Prion diseases -- Government policy -- United States : Advancing prion science : guidance for the national prion research program / Committee on Transmissible Spongiform Encephalopathies: Assessment of Relevant Science ; Rick Erdtmann and Laura B. Sivitz, editors
Prion diseases -- Research -- United States : Advancing prion science : guidance for the National Prion Research Program, interim report / Rick Erdtmann and Laura Sivitz, editors ; Committee on Transmissible Spongiform Encephalopathies: Assessment of Relevant Science, Medical Follow-Up Agency, Institute of Medicine of the National Academies
2003
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Prion Diseases -- Statistics : International Seminar on Nuclear War and Planetary Emergencies, 32nd session : the 32nd session of international seminars and international collaboration : "E. Majorana" Centre for Scientific Culture, Erice, Italy, 19-24 Aug. 2004 / edited by R. Ragaini
A group of genetic, infectious, or sporadic degenerative human and animal nervous system disorders associated with abnormal PRIONS. These diseases are characterized by conversion of the normal prion protein to an abnormal configuration via a post-translational process. In humans, these conditions generally feature DEMENTIA; ATAXIA; and a fatal outcome. Pathologic features include a spongiform encephalopathy without evidence of inflammation. The older literature occasionally refers to these as unconventional SLOW VIRUS DISEASES. (From Proc Natl Acad Sci USA 1998 Nov 10;95(23):13363-83)
A group of genetic, infectious, or sporadic degenerative human and animal nervous system disorders associated with abnormal PRIONS. These diseases are characterized by conversion of the normal prion protein to an abnormal configuration via a post-translational process. In humans, these conditions generally feature DEMENTIA; ATAXIA; and a fatal outcome. Pathologic features include a spongiform encephalopathy without evidence of inflammation. The older literature occasionally refers to these as unconventional SLOW VIRUS DISEASES. (From Proc Natl Acad Sci USA 1998 Nov 10;95(23):13363-83)
A group of genetic, infectious, or sporadic degenerative human and animal nervous system disorders associated with abnormal PRIONS. These diseases are characterized by conversion of the normal prion protein to an abnormal configuration via a post-translational process. In humans, these conditions generally feature DEMENTIA; ATAXIA; and a fatal outcome. Pathologic features include a spongiform encephalopathy without evidence of inflammation. The older literature occasionally refers to these as unconventional SLOW VIRUS DISEASES. (From Proc Natl Acad Sci USA 1998 Nov 10;95(23):13363-83)
A group of genetic, infectious, or sporadic degenerative human and animal nervous system disorders associated with abnormal PRIONS. These diseases are characterized by conversion of the normal prion protein to an abnormal configuration via a post-translational process. In humans, these conditions generally feature DEMENTIA; ATAXIA; and a fatal outcome. Pathologic features include a spongiform encephalopathy without evidence of inflammation. The older literature occasionally refers to these as unconventional SLOW VIRUS DISEASES. (From Proc Natl Acad Sci USA 1998 Nov 10;95(23):13363-83)
Prior analytics Aristotle Commentaries Early works to 1800 : David the Invincible, commentary on Aristotle's Prior analytics : critical Old Armenian text / with an English translation, introduction, and notes by Aram Topchyan ; [edited] by Aram Topchyan
Here are entered works on knowledge acquired either formally or non-formally before entrance into a specific program, usually in the area of post-secondary education